What I track —
and what it has
taught me.
I like data. Not because numbers are the point — but because they reveal things that intuition alone cannot. The most useful thing tracking has taught me is that my assumptions about my own recovery, sleep quality, and health were often wrong. My vitamin D was almost low despite living in Florida. That single data point is why I check labs. You cannot manage what you do not measure.
Data over guessing —
the case for measuring yourself.
Physicians are trained to be skeptical of n=1 data. A single patient's self-report is not evidence. Wearable data is not a clinical measurement. And yet — the alternative to tracking your own health metrics is guessing. And guessing, as it turns out, is surprisingly unreliable even for people who pay close attention.
I thought I was sleeping adequately. The data showed otherwise — deep sleep was consistently lower than I expected, and the nights I felt most rested did not always correspond to the nights the numbers looked best. I thought I was recovering well after hard weeks. The HRV trend told a different story. I thought living in Florida meant my vitamin D was fine. It was almost deficient.
None of these findings required clinical intervention. But all of them changed my behavior. That is the value of tracking at the personal level — not diagnosis, not treatment, but signal. The data points toward things worth paying attention to before they become problems.
I am also aware of the limits. A consumer wearable is not a medical device. HRV measured on an Apple Watch is not equivalent to clinical HRV measurement with validated electrodes. Sleep staging from an accelerometer is an estimate. The number I see in the morning is a proxy for the underlying physiology — useful for trends over weeks and months, not definitive for any single night. I use it as signal, not verdict.
“My vitamin D was almost low despite living in Florida year-round. That was the specific data point that convinced me that lifestyle assumptions are not a substitute for actual measurement.”
Apple Watch —
four metrics, every night.
All four tracked automatically. No manual input required beyond wearing the watch to sleep. The value is in the trend line over weeks and months — not any individual data point.
The Apple Watch is not a medical device. HRV measured on a consumer wearable uses optical sensors at the wrist rather than clinical-grade ECG electrodes — the absolute numbers are not directly comparable to clinical HRV measurements. Sleep staging from an accelerometer is an estimate, not a polysomnography. What wearables do well is track relative change in an individual over time. My HRV trending from 65 to 45 over two weeks is a meaningful signal even if neither number is clinically precise. Use the trend. Do not over-interpret any single data point.
Food tracking —
two tools, two different questions.
I track food in two distinct ways. A calorie logging app answers the question of how much I am eating. Yuka answers the question of what is actually in it. Neither replaces the other and they serve completely different purposes.
Calorie intake is easy to underestimate — significantly. On busy OR days I skip meals and eat opportunistically, which means calories land in a compressed window. On stressful days the binge eating pattern kicks in. In both cases, my intuitive sense of how much I have eaten is unreliable.
The data from the brief periods I have tracked consistently confirms this. The gap between what I thought I was eating and what I was actually eating was larger than expected. Consistently in one direction. Tracking makes that gap visible.
I am trialing calorie logging apps to find one that works on busy surgical days. Calories only — not macros, not meal timing. Get the baseline right first.
I use Yuka to scan packaged foods and supplements. Point the camera at a barcode and it pulls the ingredient list, flags additives, scores the product for ingredient quality, and identifies anything worth paying attention to.
Yuka is not perfect — its algorithm is not always consistent with peer-reviewed science. I use it as a quick filter, not a definitive verdict. The scan starts the conversation with the label. It does not end it.
Adding complexity before the habit is established increases the likelihood of abandoning it entirely. The most important tracking is the tracking that actually happens. Protein is handled through food choices — eggs, chicken, lean meat. The calorie number is what I am trying to make visible and accurate first. I will update this page when I have settled on an approach that works consistently.
Labs every few years —
what I test and why.
I get comprehensive bloodwork done periodically — not on a fixed annual schedule, but every few years or when something prompts a closer look. The goal is not to optimize every number. It is to catch things that lifestyle assumptions might be masking. The vitamin D finding is the clearest example of why this matters.
Lab reference ranges vary by laboratory, age, sex, and clinical context. A number that is flagged as low by one lab's reference range may be entirely appropriate for another individual. More importantly, interpreting bloodwork requires clinical context that a number on a page cannot provide. If you are interested in getting your own labs done, the right person to interpret them is your physician — not a webpage. What I can offer is the reasoning behind why each panel is worth checking periodically for an active, health-focused adult.
| Marker | Why it matters for active adults |
|---|---|
| Hemoglobin / Hematocrit | Oxygen-carrying capacity. Directly affects exercise performance, recovery, and fatigue. Anemia can be subtle and long-undetected. |
| WBC differential | Chronic low-grade immune activation or suppression. Useful baseline for someone managing high physical and cognitive stress loads. |
| Platelets | Clotting function. Relevant for anyone considering antiplatelet supplements (fish oil, aspirin, garlic at high doses). |
| MCV / MCH | Red cell size and hemoglobin content — can indicate iron or B12/folate deficiency before hemoglobin drops. |
| Marker | Why it matters for active adults |
|---|---|
| AST / ALT / GGT (LFTs) | Liver enzyme elevation can occur with high-dose supplements, statins, alcohol, or underlying liver pathology. Baseline is useful for anyone taking a supplement stack. |
| Creatinine / BUN | Kidney function. Creatinine supplementation modestly raises serum creatinine — knowing your baseline matters for interpretation. High protein intake also raises BUN. |
| Fasting glucose | Early insulin resistance marker. Fasting glucose can be elevated for years before diabetes develops. Lifestyle intervention works best early. |
| Electrolytes (Na, K, Cl, CO2) | Sodium, potassium, and bicarbonate balance — relevant for anyone who trains hard, sweats significantly, or uses a sauna regularly. |
| Albumin / Total protein | Nutritional status and liver synthetic function. Low albumin in an otherwise healthy active adult can indicate inadequate protein intake. |
| Marker | Why it matters for active adults |
|---|---|
| Serum Mg | Proxy for magnesium status. Affected by heavy sweating, sauna use, high caffeine intake, and GI absorption. Low magnesium affects sleep quality, muscle recovery, and energy metabolism. |
| Marker | Why it matters for active adults |
|---|---|
| Serum phosphorus | Bone mineral metabolism alongside calcium and vitamin D. Low phosphorus can indicate malabsorption or inadequate intake; elevated levels can reflect kidney dysfunction. |
| Marker | Why it matters for active adults |
|---|---|
| Total testosterone | Overall androgen status. Affected by sleep quality, body composition, stress, and training load — all of which are tracked independently. |
| Free testosterone | The biologically active fraction. More informative than total testosterone alone, particularly in older men with elevated SHBG. |
| SHBG | Sex hormone binding globulin — determines what fraction of total testosterone is free and active. Increases with age. |
| Marker | Why it matters for active adults |
|---|---|
| TSH | Primary thyroid screen. Elevated TSH signals hypothyroidism; suppressed TSH signals hyperthyroidism. Symptoms of both mimic common lifestyle complaints. |
| Free T4 / Free T3 (if TSH abnormal) | More detailed thyroid assessment when TSH is out of range. Not routinely checked when TSH is normal. |
| Marker | Why it matters for active adults |
|---|---|
| Serum B12 | Neurological function, cognitive performance, red cell synthesis. Deficiency develops slowly and can cause irreversible neurological damage if missed. |
| Marker | Why it matters for active adults |
|---|---|
| Serum folate | DNA synthesis and homocysteine regulation. Interacts closely with B12 — both should be checked together. |
| Homocysteine (if low folate/B12) | Elevated homocysteine is an independent cardiovascular and neurological risk factor — a downstream consequence of inadequate B12 or folate. |
| Marker | Why it matters for active adults |
|---|---|
| HbA1c | 3-month glucose average. More reliable than fasting glucose for detecting early insulin resistance. Pre-diabetes is asymptomatic and reversible — if caught early. |
| Marker | Why it matters for active adults |
|---|---|
| ESR | Broad inflammatory screen. Non-specific but sensitive. Elevation in an asymptomatic person warrants further workup for infection, autoimmune disease, or occult malignancy. |
| CRP (often checked alongside) | C-reactive protein — a more specific and rapidly responding inflammatory marker than ESR. Together they provide a fuller picture of systemic inflammatory status. |
| Marker | Why it matters for active adults |
|---|---|
| LDL cholesterol | Primary atherogenic lipoprotein. Standard LDL-C is an estimate — LDL-P (particle number) or ApoB are more precise for risk stratification. |
| HDL cholesterol | Cardioprotective. Raised by regular aerobic exercise. Low HDL is an independent cardiovascular risk factor. |
| Triglycerides | Highly responsive to diet and lifestyle. Elevated triglycerides with low HDL is the classic insulin resistance lipid pattern. |
| Total cholesterol / TC:HDL ratio | Broad cardiovascular risk index. Less informative than LDL-P or ApoB but widely reported and useful for trend tracking. |
| ApoB (if available) | Counts all atherogenic particles directly — more precise than calculated LDL-C for cardiovascular risk stratification. |
| Marker | Why it matters for active adults |
|---|---|
| 25-OH Vitamin D | The storage form of vitamin D and the correct test to order for status assessment. Supports bone density, immune function, muscle performance, and mood. Deficiency is common even in sunny climates for people who spend most of the day indoors. |
Track the trend.
Check the labs.
Do not assume.
The vitamin D finding is the reason. You cannot manage what you do not measure — and you cannot measure what you do not test. If your spine is limiting what you can track, train, or do, that is worth addressing.
Request a Spine Consultation